10: A patient goes to the clinic with symptoms of muscle spasm, hallucinations, weak and brittle bones. Tests showed decreased levels of calcitriol in blood but higher levels of calcitonin. Explain the cause of symptoms observed and how the altered levels of the two hormones mentioned might have been the cause.
9. In a slow progressive disease in humans, muscle function is lost around 35 years of age. Design experiments to identify the molecular players involved in the disease development and how you will try to find the cure for it.
7. Duchenne muscular dystrophy (DMD) results from a mutation in Dystrophin gene. Can you treat the disease by injecting recombinant form dystrophin protein? Explain!
5. Why does a skeletal muscle twitch last longer
than a skeletal muscle action potential?
4. Describe the renal regulatory pathway that
controls the permeability of the nephron’s collecting duct.
3. A particular mammalian tissue suddenly
exhibits increased oxygen consumption. List the
physiological mechanisms that will help in fulfilling the
tissue’s oxygen demand. Integrate the concepts of
cardiovascular physiology and gas transport to provide a
complete response.
How does the presence of cristate in the inner mitochondrial membrane increase the productivity of aerobic respiration in eukaryocytes?
Why is it that prokaryotic cells gain more ATPs from glucose as compared to eukaryotic cells?
What is the gross number of ATP molecules produced from one glucose molecule during aerobic respiration?
How many ATPs are produced by the respiratory chain from all the molecules of FADH2 formed from the complete oxidation of one glucose molecule?