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Let's tackle a couple of autoimmune diseases (diseases where our own body attacks itself) that impact the neuromuscular junction (NMJ). The Lambert-Eaton Syndrome—an autoimmune disease marked by the destruction of presynaptic Ca2+ channels—and Myasthenia Gravis—an autoimmune disease marked by Acetylcholine (ACh) receptor destruction. What areas of the NMJ are affected by these two diseases? What is an appropriate treatment for these two diseases? Would more ACh help? Would more Ca2+ help? Would more AChE help?
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