Answer to Question #200975 in Psychology for Thobile

Question

Misty Mayer, works as the receptionist in a private hospital. She is known to be a friendly, well-balanced and optimistic person with diverse hobbies and good interpersonal relations. However, since a month ago Misty has succumbed to severe depression, without any change in her work and life circumstances. The psychiatrist prescribes an antidepressant to better regulate her mood and her sleeping patterns. A deficit in which neurotransmitter may be the cause of Misty’s depression?

Answer 1

Introduction

Synaptic stress is the interest-dependent form of temporary synaptic liability that results in the small-term incapacity of the neurons to fire and hence transact a load of the signal. It is imagined to be a form of adverse outcomes to restrain control specific structure of the nervous system. Synaptic stress causes a short-term depletion of the synaptic vesicles that host the neurotransmission in the synapse broadly release by tenacity high recurrence neural stimulation. This essay elaborates on synaptic stress and how this affects the health and ability to work for individuals such as Misty.

 The synapse produces the neurotransmitters to regulate the signal to the postsynaptic cell.it has also experimented that the synaptic tiredness could be caused by postsynaptic receptors affectionless or substitute in postsynaptic inactiveness transmission. Still, current analysis has encouraged that it is mainly a presynaptic phenomenon (Dorninger et al.,2019). Synaptic tiredness can influence a lot of many cells of varying neurons. Antistress has a temporary and permanent effect on stressed patients (Wan et al.,2020). The transient effects are elaborated in the analysis that explains that the stress is cutely introduced by an instant reduction in the catecholamines in the brain.

 Antistress has a role of instantly possess this reduction and revival of the expected levels of neurotransmitters in the brain (Wan et al., 2020). Under depression states, vesicle exocytosis is prohibited, and the production of catecholamines results in the stress of presynaptic cells because of depleted neurtransimmted (Martino & Magioncalda., 2021)..Therapeutic doses of fluoxetine have been illustrated to reduce these neuronal tiredness conditions by inheriting vesicle produced and minimizing synaptic tiredness in the hippocampal neuron. Most of the analysis portrays that the fluoxetine as well as existing antidepressants that have a role through all standard apparatus as fluoxetine increases neuro recovery and neurotransmission to minimize the chance of stress.

Dopamine is a fluid found naturally in the body of a human being. It has the role of hosting this neurotransmitter, propagating signals from the body to the brain through vesicles. It has the function of managing the propagations of an individual and their emotional responses ( Wan et al.,2020). The right balance of this chemical dopamine is crucial for an individual's physical and mental well-being. The critical role of the brain, such as mood, sleep, memory learning, focuses for an extended period. And motor management is affected by the levels of this chemical dopamine. The lack of this chemical dopamine may be compared with the specific medical state that involves depression and Parkinson disease (Martino & Magioncalda.,2021).

Dopamine shortage can result in a decrease in the quantity of dopamine manufactured by the body or a challenge with the receptors in the brain. The signs of dopamine shortage rely on the underlying effects. For illustration, an individual with the condition of Parkinson disease will encounter wildly varying signs and symptoms from the individual with less dopamine due to drugs utilized. Some of the existing characters of the shortages are muscle cramps, spasms, aches, loss of balance, constipation, challenges in eating and swallowing, loss of weight: soft focus or concentration, suicidal thoughts, and low self-awareness (Martino & Magioncalda.,2021). Misty Mayer has Parkinson's disease. Therefore the prescription of antidepressants will enable Misty Mayer to be in a position to control depression.

References

Wan, L., Ge, W. R., Zhang, S., Sun, Y. L., Wang, B., & Yang, G. (2020). Case-control study of the effects of gut microbiota composition on neurotransmitter metabolic pathways in children with attention deficit hyperactivity disorder. Frontiers in neuroscience, 14, 127.

Dorninger, F., König, T., Scholze, P., Berger, M. L., Zeitler, G., Wiesinger, C., ... & Berger, J. (2019). It disturbed neurotransmitter homeostasis in ether lipid deficiency. Human molecular genetics, 28(12), 2046-2061.

Martino, M., & Magioncalda, P. (2021). Tracing the psychopathology of bipolar disorder to the functional architecture of intrinsic brain activity and its neurotransmitter modulation: a three-dimensional model. Molecular Psychiatry, 1-10.