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There are three histologic specimens of cartilaginous tissue. Two of them are stained with hematoxylin-eosin, and one histologic specimen is stained with orcein. What fibres and in what type of cartilaginous tissue can be determined by such dyes? What functional properties of the cartilaginous tissue do they cause?


The cells of an ant and a horse are roughly the same size; a horse is bigger simply because it has more. What is the main benefit for cells to be very small?




What can cells do to increase their Surface Area -to- Volume Ratio?




Changing the shape of a protein is also known as a change in its ___________.

Answer choices

a) conformation

b) primary structure

c) nature


Changing the shape of a protein is also known as a change in its ___________.


Answer all of the following questions concerning protein synthesis by the endoplasmic reticulum: (Total of 7 Marks) a. Name the structure that binds to the SRP at the honest of secretory protein synthesis. (1 Mark) b What happens to protein synthesis when the above binding (a) is established on the nascent polypeptide chain? (1 Mark) c What facilitates the association of the ribosome with the translocon of the ER membrane? (2 Mark) d. Which molecule recognizes and binds to unfolded or misfolded proteins and help them attain their native structure. (1 Mark) e. Which molecule is responsible for degrading misfolded proteins? (1 Mark) t. Where are misfolded secretory proteins eventually destroyed? (1 Mark)


generally, is glycolysis endergonic or exergonic?


Explain what is meant by the term ‘linked genes’ with respect to homologous recombination.


Question 5: Which organ and cell type are primarily affected by the mutation? Is this consistent with the symptoms observed in SCA1 patients?

Mice are generally a good model for human diseases, if you wish, you can read more about the benefits of the mouse model on The Jackson Laboratory website  <span style="color:#E90167">Advantages of the mouse as a model organism</span>.

Mouse models of SCA1 have been developed and helped researchers to understand this disease and to experiment with new treatments.



The Ras protein functions as a molecular switch that is set to its “on” state by other proteins that cause it to release its bound GDP and bind GTP. A GTPase-activating protein helps reset the switch to the “off” state by inducing Ras to hydrolyze its bound GTP to GDP much more rapidly than it would without this encouragement. Thus, Ras Works like a light switch that one person turns on and another turns off. You are studying a mutant cell that lacks the GTPase-activating protein. What abnormalities would you expect to find in the way in which Ras activity responds to extracellular signals?

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